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Product Details
CAS No.: 15366-32-2
Formula: C3h7no2
EINECS: 203-538-6
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  • Premium Creatine Ethyl Ester HCl for Enhanced Performance
  • Premium Creatine Ethyl Ester HCl for Enhanced Performance
  • Premium Creatine Ethyl Ester HCl for Enhanced Performance
  • Premium Creatine Ethyl Ester HCl for Enhanced Performance
  • Premium Creatine Ethyl Ester HCl for Enhanced Performance
  • Premium Creatine Ethyl Ester HCl for Enhanced Performance
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Basic Info.

Model NO.
JT-474
Resource
Chemosynthesis
Transport Package
25kgs/Drum or Carton, Inner by Aluminum Foil Bag
Specification
99%
Trademark
JT
Origin
China
HS Code
1302199099
Production Capacity
1000kg/Month

Product Description

Premium Creatine Ethyl Ester HCl for Enhanced Performance
Product name Creatine ethylester HCL
Appearance White powder
CAS 15366-32-2
Test Method HPLC
Chemical Formula C6H14ClN3O2
Molecular Weight
89.09
Creatine ethyl ester hydrochloride, with a CAS number of 15366-32-2, has a molecular formula of C6H14ClN3O2 and a molecular weight of 195.647. It is a white crystalline powder with hygroscopicity and a melting point of 162-165°C [4]. This substance is mainly used as a food additive in nutrition, widely applied in the preparation of drugs for treating heart diseases and respiratory insufficiency, and also used in the formulation of health foods with anti-aging effects (such as creatine citrate, creatine malate, etc.).
Specification:  99%
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Improving respiratory function
NAC can stimulate the secretion and formation of surfactant by type II cells in the alveoli, enabling the alveoli to expand effectively and increasing the area for gas exchange. It achieves this by activating anti-protease and inhibiting the elastase released by polymorphonuclear neutrophils that damages alveolar elastin. A group of studies have shown that compared with the non-treatment group, the annual average attenuation of FEV1 in the treatment group using NAC was significantly lower.
Antioxidant effect
The free radicals derived from oxidative processes play an important role in the pathophysiology of the lungs. Smoking or the release of endogenous substances from inflammatory cells (neutrophils or macrophages) can lead to an increase in free radicals. The inflammatory response in the lungs can produce a large amount of oxygen free radicals and elastase, which destroy the activity of α1-proteinase inhibitor and cause damage to lung tissue, resulting in the destruction of alveolar structure. Free radicals promote the development of emphysema by affecting the balance between elastase and anti-elastase. One of the main systems of antioxidants is the glutathione (GSH) redox cycle and the increase in antioxidant protection.
Enhancing the body's immune function
NAC can inhibit the replication of viruses, stimulate the replication response of T-lymphocytes, increase the number and activity of T-lymphocyte subsets, increase the adhesion and phagocytic function of macrophages, protect cells from damage caused by viral infection, and thus play a role in preventing and reducing respiratory infections.
Reducing the number of bacteria in the airways
The increase in the deterioration rate of patients with chronic bronchitis may be related to the change in the bacterial flora in the bronchi or immune deficiency. A study (EurRespir J. 1994;7;94-101) using bronchoscopy and a protective sputum brush to collect lower respiratory tract sputum for bacterial culture showed that in 21 patients in the non-NAC treatment group, 15 types of bacteria were isolated, while in 16 patients treated with NAC, only 3 types of bacteria were isolated. This provides a reasonable explanation for the reduction in the deterioration rate of chronic bronchitis patients taking NAC.
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