Quality Health Nicotinamide Riboside Chloride Food Grade Support DNA Repair

• CAS No.: 1341-23-7
• Formula: C11h15n2o5
• EINECS: 130-626-2
• Model NO.: JT-270
• Resource: Chemosynthesis
• Molecular Weight: 256.26
• Test Method: HPLC
• Water Solubility: Soluble to 0.70 Mg/Ml in Water
• Transport Package: 25kgs/Drum or Carton, Inner by Aluminum Foil Bag
• Specification: 99%
• Origin: China
• HS Code: 1302199099
• Production Capacity: 1000kg/Month

Product Description

Product name	Nicotinamide Riboside
Appearance	White powder
CAS	1341-23-7
Molecular Formula	C11H16N2O5
Molecular Weight	256.26
Synthetic Method	Nicotinamide +β -D-furanribose -1,2,3, 5-tetraacetate → nicotinamide ribose

Nicotinamide Riboside
Nicotinamide riboside (NR) is a derivative of vitamin B3 and a precursor substrate of Naga factor nicotinamide adenine dinucleotide (NAD+). It can increase NAD+ levels after intake. It is a white or light yellow crystalline powder, odorless, bitter in taste, and slightly hygroscopic.
Specification:  99%
Nicotinamide Riboside (NR) is a derivative of vitamin B3 and participates in cellular energy metabolism and repair as a precursor of NAD+ (coenzyme I). It supports mitochondrial function, delays aging and improves metabolism by increasing NAD+ levels in the body. However, the specific effects vary from person to person and need to be evaluated in combination with an individual's health status.
Mechanism of Action and Core Functions
1.Promote NAD+ generation
NAD+ is a key coenzyme in cellular energy metabolism, and its level gradually decreases with age. NR is converted into NAD+ after being absorbed through the intestine, which helps maintain the energy balance of cells and enhances mitochondrial function.
2.Support DNA repair
NAD+ activates the Sirtuins family of longevity proteins, participating in DNA repair and oxidative stress regulation, which may delay age-related functional decline.
3.Improving Metabolic Health
Animal experiments have shown that NR may improve  sensitivity and reduce blood lipids, but the clinical evidence for human metabolic diseases (such as diabetes) is limited.